NUR 631 Topic 3 Assignment Task: CLC – Acid-Base Case Study PowerPoint (2 Versions)

NUR 631 Topic 3 Assignment Task: CLC – Acid-Base Case Study PowerPoint (2 Versions)

(NUR 631 Topic 3 Assignment)

This is a Collaborative Learning Community (CLC) assignment.

Working in teams and collaboration is an essential skill that is prevalent in clinical situations. In this assignment, you will be working in a group to create a PowerPoint to address the “Acid-Base Case Study. ” The instructor will divide the class into CLC groups. The PowerPoint should be 10-15 slides and contain speaker notes. Each student is responsible for participation within the CLC group. The instructor will monitor the CLC Discussion Forum. One member of the group will submit the assignment to the instructor. Each of the following should … addressed:

Permalink: https://collepals.com/nur-631-topic-3-…point-2-versions/ ‎

1. Research and fill in the normal values for the table in the “Acid-Base Case Study” resource. Include the table in your PowerPoint.
2. What type of acid-base disturbance is Mr. Davis suffering from? Why?
3. What role does excessive alcohol consumption play in the acid-base disturbance seen?
4. What type of fluid or electrolyte imbalances does Mr. Davis have and why?
5. Calculate the anion gap. Is it high or normal? Why is it high or normal? What information does the anion gap give the provider?
6. Are Mr. Davis’ respiratory and renal systems attempting to compensate for his acid-base disturbance? If so, how are they compensating and what evidence do you have that they are compensating?
7. Explain the rationale for the low glucose level and high urine ketones.
8. Is the protein level seen in the UA abnormal? Provide a rationale. How do the findings relate to Mr. Davis?

While APA style is not required for the body of this assignment, solid academic writing is expected, and in-text citations and references should be presented using APA documentation guidelines, which can be found in the APA Style Guide, located in the Student Success Center.

You are required to cite three to five sources to complete this assignment. Sources must … published within the last 5 years and appropriate for the assignment criteria and nursing content.

This assignment uses a rubric. Please review the rubric prior to beginning the assignment to become familiar with the expectations for successful completion.

ADDITIONAL INFORMATION;

What role does excessive alcohol consumption play in the acid-base disturbance?

Introduction

Alcoholic ketoacidosis (AKA, “acidosis”) is a serious complication of excessive alcohol consumption, which can result in death. Whereas it’s most commonly seen in patients who have been drinking for days or even weeks at a time, it also occurs after an acute binge episode when the patient is sober but still has excess acetaldehyde in their system. Acetaldehyde is one of the byproducts produced during alcohol metabolism and builds up over time as ethanol continues to be consumed. The presence of high levels of acetaldehyde can cause metabolic acidosis, which can lead to injury or death if untreated.

Introduction

In this article, we will discuss the metabolic acidosis caused by alcohol in the body. We will also look at how alcohol affects the body’s acid-base balance and what role it plays in this disturbance.

The main function of the kidneys is to maintain a stable balance of chemicals in the body. These include water, minerals and acids. There are three types of acids:

organic acids, lactic acid and metabolic acids. Metabolic acidosis is the most common form of kidney disease. It occurs when there is too much acid in the body, which can lead to serious health problems.

Background

Alcohol is a toxin. Alcoholic ketoacidosis (AKA) is a life-threatening condition that occurs when the body cannot metabolize alcohol and it produces toxic byproducts that poison the body. The liver tries to metabolize ethanol, but it creates toxic metabolites instead of energy-producing glucose molecules. When this happens, your blood becomes acidic and bile acids are released into your bloodstream causing nausea and vomiting as well as causing heartburn or a feeling of being full soon after eating food.

In contrast to AKA, alcoholic metabolic acidosis (AMA) refers to low pH levels in the blood due to excess production of acetate by liver cells while consuming large amounts of carbohydrates/sugars through drinks like beer or wine

Assessment of acid-base disturbance

• How to assess acid-base disturbance?

• Use of laboratory tests: A blood sample is taken from the patient, and it is analyzed in the laboratory. The results will help determine whether there are any abnormalities in the pH level or base excess/deficit (BE). If a BE has occurred, this can be confirmed with a urine test that measures how much bicarbonate is excreted into urine over time.

• Clinical signs: The clinical signs are based on how your body responds to changes in acid-base levels; for example, if you have low blood volume and increased breathing rate as well as feeling dizzy then this would indicate an oxygen deficit due to low CO2 levels from not enough ventilation (CO2 being released from cells).

Alcoholic ketoacidosis (acute)

Alcoholic ketoacidosis (AK) is a type of metabolic acidosis caused by alcohol consumption. In AK, the body produces excess amounts of ketones when it breaks down fat for energy.

Ketones are produced when the body breaks down fat for energy. Ketone bodies are usually excreted in urine and bile, but they can also accumulate in the blood stream if you have not enough insulin production or storage capacity to use them as an alternate source of fuel. If this occurs over a prolonged period (several days), your kidneys will start to fail and you’ll go into cardiac arrest because there won’t be enough oxygenated blood reaching your organs; this condition is known as septicemia or sepsis from Septicemia = Sepsis

Alcoholic metabolic acidosis (chronic)

Acute alcoholic ketoacidosis, which is characterized by a high level of ketones in the blood, is a medical emergency. It occurs when alcohol consumption results in low levels of insulin (in response to excess calories), leading to tissue damage from lack of oxygen and nutrients. When this happens, children are at risk for coma or death because their bodies cannot use glucose efficiently for energy.

Chronic alcoholic metabolic acidosis involves excessive intake of alcohol over time with no food intake at all and may result in organ damage such as liver cirrhosis or pancreatitis if left untreated

Metabolic acidosis can be seen in patients who consume excessive alcohol.

Metabolic acidosis can be seen in patients who consume excessive alcohol. The metabolic acidosis is a result of the breakdown of alcohol and other substances, such as acetaldehyde and ketone bodies. These substances are formed when you take in carbohydrates (carbohydrates) and break them down into energy.

The process begins with your body burning food for energy, which produces carbon dioxide (CO2). The CO2 leaves your body as you breathe out; this is why you exhale air with every breath you take! In order to create more CO2 from carbohydrates or fat deposits in your body, they must be burned first before they can be turned into CO2 by combining with oxygen molecules through photosynthesis known as respiration.

Conclusion

We hope that this article has helped you understand more about the role of alcohol in metabolic acidosis. As we’ve seen, both acute and chronic situations are possible, but fortunately there are ways to prevent them. If you’re concerned about your own health or the health of a loved one who might be suffering from an alcohol-induced acidosis, make sure they see their doctor immediately. The sooner they receive treatment for the condition, the better chance they’ll have at surviving it with minimal damage to their organs and tissues.