NUR 631 Topic 12 Discussion Questions with Answers
NUR 631 Topic 12 Discussion Questions with Answers
NUR 631 Topic 12 Discussion Question 1
Select two of the following discussion questions for your discussion response. Indicate which questions you have chosen using the format displayed in the “Discussion Forum Sample.”
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Explain the concept of azotemia (including prerenal, renal, and postrenal), causes, and diagnostic measures used to identify each.
Spinal cord injuries carry a high risk of neurogenic bladder and reflex incontinence. Explain the mechanisms responsible for these disorders.
NUR 631 Topic 12 Discussion Question 2
Select two of the following discussion questions for your discussion response. Indicate which questions you have chosen using the format displayed in the “Discussion Forum Sample.”
Explain the differences between lower tract urinary symptoms and erectile disorder.
A patient presents with flank pain. You suspect renal calculi. What is the pathophysiological reason for development of renal calculi and associated treatment considerations to rid renal stones and prevent future development of others?
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ADDITIONAL INFORMATION;
Explain the concept of azotemia
Introduction
Azotemia is a term used to describe nitrogenous waste products accumulating in the blood. The word ‘azotemia’ comes from Greek words ‘azotos’meaning nitrogen, and ‘haima’ meaning blood. It was first used in 1843 by the German pathologist, Carl von Grobben.
Section: Derived from Greek words ‘azotos’meaning nitrogen, and ‘haima’ meaning blood. A person with elevated levels of BUN (creatinine) or creatinine will have azotemia if there are signs of renal disease such as fever, nausea/vomiting or diarrhea
Azotemia is defined as nitrogenous waste products accumulating in the blood.
Azotemia is a common finding in patients with kidney disease. It’s also considered to be a serious medical condition that can lead to death.
Azotemia refers to the presence of nitrogenous waste products in the blood and occurs when there isn’t enough oxygen-rich red blood cells circulating through your bloodstream, causing them to break down and release nitrogenous materials into your body’s fluids.
This condition can lead to serious injury or even death. The most common cause of azotemia is kidney failure, although it can also be caused by other conditions that affect the liver, heart or lungs.
This term was first used in 1843 by the German pathologist, Carl von Grobben.
The term azotemia was first used by the German pathologist, Carl von Grobben. He described this condition in 1843 and used it to describe a patient with kidney disease. This term is still used today to describe patients who have high levels of nitrogenous waste in their blood and urine.
The condition azotemia is most commonly associated with kidney disease. This condition can occur in people who have had their kidneys removed or who have a metabolic disorder that prevents them from removing nitrogenous wastes from their blood.
Derived from Greek words ‘azotos’meaning nitrogen, and ‘haima’ meaning blood.
Azotemia is a medical condition in which there is an excess of nitrogen in the blood, resulting in high levels of urea. This can be caused by various factors, including:
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Sickle-cell disease or other blood disorders (such as thalassemia) that cause red blood cells to change shape and form into spikes that damage tissue around them
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High levels of proteinuria caused by kidney problems or liver disease
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Low levels of hemoglobin in the bloodstream due to poor oxygen transport
Blood urea nitrogen (BUN) and creatinine are two clinical markers of azotemia.
Blood urea nitrogen (BUN) is a waste product of protein metabolism. It is produced by the liver and filtered by the kidneys, where it combines with other compounds in urine. BUN levels are an indirect measure of kidney function; higher levels are associated with azotemia, while lower levels suggest pre-renal failure or some form of obstruction that prevents excretion from the kidneys.
Many conditions lead to azotemia including kidney disease, dehydration, shock, heart failure, liver disease and cancer.
Azotemia can be caused by a number of different conditions, including kidney disease, dehydration and shock.
Other causes include drug toxicity, burns and crush injuries.
Symptoms of hyponatremia include nausea and vomiting, headache, lethargy, confusion and muscle cramps. Severe cases can lead to seizures, coma and even death.
Renal and non-renal causes both can produce an abnormally high or low BUN or creatinine.
Renal causes of azotemia include acute kidney injury (AKI), chronic kidney disease (CKD), and kidney failure. Non-renal causes include dehydration, shock, heart failure, liver disease and cancer.
The most common cause of azotemia is kidney failure. When the kidneys stop working properly, they can’t remove waste products from the body fast enough. The buildup of these wastes can lead to an increase in urea and creatinine levels in your blood.
One of the most common causes of azotemia is acute kidney injury (AKI).
One of the most common causes of azotemia is acute kidney injury (AKI). AKI, also known as acute renal failure, is a serious medical condition that can lead to death. The condition can be caused by many different factors including infections, dehydration and sepsis.
AKI may be defined as an increase in BUN and creatinine levels. In order for someone with AKI to develop chronic kidney disease (CKD), their kidneys must fail permanently or deteriorate significantly enough so that they no longer function properly.
Conclusion
The most common cause of azotemia is acute kidney injury (AKI). AKI is a medical emergency that occurs when the kidneys fail to adequately remove waste products from the blood. There are several different types of AKI, but they all result in excess nitrogenous waste in the bloodstream. The most common causes of this disease include dehydration or shock; however, it can also be caused by other conditions such as cancer or hepatitis B virus infection.
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